Hyperhidrosis Homeopathic Cure
The viscera, when they are healthy, give rise to minimal conscious sensation. Fullness, gurgles, and a sensation of gas are the consequences of ingestion or sources of a need for elimination. In day-to-day activities, these sensations often increase to levels of mild discomfort, but when viscera become diseased or inflamed, the same stimuli that produce innocuous sensations can become an overwhelming source of sensations that can stop all activity and can demand complete attention. Nausea occurs commonly with visceral pains as do other autonomic responses such as sweating to the point of diaphoresis, piloerection, and dyspnea. It is clinical lore that visceral pains produce strong emotional responses to the point that they may appear out of proportion to the perceived intensity of the pain. Strong emotions are not only evoked by visceral sensations but also serve to evoke further visceral sensations such that a positive
Features Fight or flight hypertension, tachycardia, sweating, fever, excitation-psychomotor agitation, tremor, seizures, dilated pupils. Causes Amphetamines diet drugs, cocaine, theophylline, caffeine, methylphenidate, mono-amine oxidase inhibitors over-the-counter cold medications, especially those containing phenylpropanolamine (PPA), ephedrine, and pseudoephedrine.
Symptoms of hypoglycaemia were first reported in relation to tumours of the pancreas (Wilder 1927). As early as 1927, the symptoms of hypoglycaemia were recognised as forming two groups the first occurring during mild reactions comprising anxiety, weakness, sweating, hunger, tremor and palpitations and the second more severe group including mood changes, speech and visual disturbances, drowsiness, convulsions and coma (Harrop 1927). It was also noted that some patients did not experience the usual symptoms of hypoglycaemia until their blood glucose had reached much lower concentrations (Lawrence 1941). Symptom profiles provoked by hypoglycaemia are idiosyncratic and vary in character, pattern and intensity between individuals and even within individuals over time (Pennebaker et al. 1981). Sweating
Important types of information (1) information about the feared stimuli or situation (2) information about the person's response to the feared stimuli or situation and (3) information about the meaning of the feared stimuli and the consequent response. Foa and Kozak (1986) posited that the fear networks of individuals with PTSD differ from the fear networks of individuals with other anxiety disorders in three ways. First, the fear network of individuals with PTSD is larger because it contains a greater number of erroneous or inaccurate connections between stimulus, response, and meaning elements. Second, the network is more easily activated by stimulus, response, or meaning elements. Third, the affective and physiological response elements of the networks are more intense. Accordingly, for individuals with PTSD, stimuli reminiscent of the traumatic experience activate the fear network and prompt states of high sympathetic arousal (e.g., increased heart rate and blood pressure,...
Weight loss in PCC patients is usual, although obesity cannot exclude the diagnosis. The weight reduction is partly due to increased metabolic rate, excessive sweating, and heat intolerance. Fever may also be present 31-33 . Weight loss is sustained by an activation of lipolysis in white adipose tissue. An activation of brown fat is also evident in patients with PCC 34 . It is noteworthy that, while adipose tissue constitutes the bulk of body fat stores and primarily has as an energy storage function, brown adipose tissue functions principally to generate heat in humans and many other species 34 .
Vectors All pufferfish (balloonfish, blowfish, fugu fish, globefish, swellfish, toadfish), porcupine fish, marine sunfish xanthid crabs, marine worms blue-ringed octopus bites skin secretions of some newts, frogs, and toads. Incubation 10-20 minutes. Symptoms Initial paresthesias, perioral burning, then salivation, headache, nausea and vomiting (diarrhea rare), sweating, glove and stocking paresthesias then numbness, tremor, ataxia, dysarthria, dysphagia, respiratory depression then paralysis, cardiovascular instability, stupor, and coma. Diagnosis Mouse bioassay, TLC, HPLC, gas chromatography mass spectrometry. Treatment Supportive protect airway, gastric lavage then AC-MDAC, IV fluids, vasopressors, and mechanical ventilation. Prognosis CFR 62 survivors will recover within 1 week of ICU care (not universally available, especially in developing world).
Other aspects of MPS may be explained by sympathetically maintained pain, including painful skin rolling, hypersensitivity of the skin and muscles to touch and pressure, vasomotor changes including pallor, hyperemia, subjective coldness, and hyperhidrosis, and the marked central and neuropharmacological reactions to emotional stress. Clinically speaking, how can the diagnoses of these two entities be differentiated First, on examination, the clinician may find painful skin rolling, dermato-graphia, and hyperhidrosis, particularly on sudomotor or sweat testing, and, on the face, scleral injection, lacrimation, ipsilateral rhinorrhea, even a partial Horner's syndrome, with ptosis and meiosis, but not anhidrosis. Further examination of the skin may reveal flushing over the painful areas, with associated warmth and extreme sensitivity to palpation.
SLUDE Muscarinic features Salivation, Lacrimation, Urination, Defecation, Emesis, plus miosis, bronchorrhea and broncho-spasm DUMBBELS Diarrhea, Urination, Miosis, Bronchorrhea, Bronchospasm, Emesis, Lacrimation, and Salivation. Nicotinic features Weakness, fascicula-tions, sweating, tachycardia, hypertension.
Glaucoma and the constricted pupils of the morphine addict are well known if not so commonly seen. When a constricted pupil on one side is observed it is important to note the position of the eyelids. A slight degree of associated ptosis indicates the possibility of Horner's syndrome. The total syndrome comprises miosis, narrowing of the palpebral fissure because of paralysis of the smooth muscle in the eyelids (Muller's muscle), loss of sweating over the affected side of the forehead, a slight reduction of the intraocular pressure and enophthalmus (sunken globe). Horner's syndrome can be caused by a wide diversity of lesions anywhere along the sympathetic pathway. While a Pancoast's apical lung tumour is classically associated with Horner's syndrome, it is quite often noted in the elderly as an isolated finding and investigation fails to reveal a cause. The Argyll Robertson (AR) pupil is a rare but famous example of the miosed pupil, which responds to accommodation but not to direct...
CRPS is characterized by chronic pain, usually in a limb, associated with local changes in sweating, skin color, skin sensitivity, skin temperature, swelling, muscle spasm, wasting of muscles, and changes in hair and nail growth. Patients do not necessarily present with all the symptoms and signs.
The clinical use of stimulants in narcolepsy has been the object of an American Sleep Disorders Association (ASDA) Standards of Practice publication. Typically, the patient is started at a low dose, which is then increased progressively to obtain satisfactory results. This final dose varies widely from patient to patient. In adults, methylphenidate and amphetamines at dosages of more than 60 mg day do not significantly improve EDS without the appearance of long-term side effects, including frequent worsening of the nocturnal sleep disruption. The drug is usually administered in three divided doses with a maximum of 20 mg in the morning, 20 mg at lunchtime, and 20 mg at 3 pm - never later. Therefore, short naps are necessary. The combination of pharmacological agents and two short naps provides the best daily response to EDS, with no stimulant drug taken after 3 pm. The slow-release form may provide gradual and delayed response during the daytime. Side effects such as headaches,...
A 56-yr-old Russian woman with a history of hypertension went to an emergency room (ER) with a severe headache that was unrelieved by over-the-counter analgesics. Ten years prior to this, while living in the Ukraine, she began experiencing intense throbbing bifrontal headaches that occurred two to three times a week. The headaches were accompanied by visual changes, pallor, and generalized weakness. Over the next 5 yr, the headaches became more severe and were accompanied by symptoms of sweating and palpitations. A few months prior to the ER visit, she sought medical attention when the headaches began to disturb her on a daily basis. Her blood pressure was noted to be elevated, and she started treatment with hydrochlorthiazide and methyldopa. Although her blood pressure apparently normalized with this therapy, she continued to experience heat intolerance. One week prior to her ER visit, she was given pseudoephedrine for a sinus headache.
The most common complaints found in hyperthyroidism include muscle weakness and pain, TrPs, heat intolerance, increased sweating, thinning hair, increased appetite, emotional mental difficulties, and sexual dysfunction. The physical findings may reveal a goiter, proptosis, loss of convergence, lid lag, increased deep tendon reflexes, tachycardia, cardiac arrhythmias, and a fine, fast tremor of the hands fingers. Laboratory findings include high levels of T3 (triiodo-thyronine), T4, and free thyroxine index. The TSH is typically low.
In contrast to uncomplicated starvation, where the energy expenditure is decreased as compensation, an elevated metabolic rate was recognised. Several symptoms such as tachycardia, hyperp-noea, sweating and a rise in body temperature indicated an increase of the metabolic rate that was in sharp contrast to the reduced energy supply in these patients 25 . In 1916, the increase in the basal metabolic rate was directly documented 26, 27 . Increased metabolic demands of several specific tissues were discussed as one underlying reason for this finding. Decreased efficiency of the respiratory system due to reduced compliance 28 and capacity of the lungs, together with hyperventilation, result in higher energy demands of the respiratory muscles 29 . In the case of patients with congestive heart failure it was also suggested that the hypertrophic myocardium may contribute to the hypermetabolism in chronic heart failure 30, 31 . The combination of an increase in total energy consumption of the...
Based on early observations of sweating, tachycardia, venoconstriction and systemic increased vascular resistance with reduced cutaneous and renal blood flow, an overactivity of the sympathetic nervous system was recognised. The finding of high norepinephrine levels in chronic heart failure patients by Chidsey, Harrison and Braunwald in 1962 supported this hypothesis 15 . The authors recognised this as a compensatory mechanism to improve cardiac performance.
DTs are most likely to develop if the patient has had an alcohol withdrawal seizure or a concomitant medical disorder, such as an infection, hepatic insufficiency, pancreatitis, subdural hematoma, or a bone fracture. Onset is usually 2-3 days after cessation of alcohol use and usually lasts 3-7 days, but can be prolonged. DTs must be considered a medical emergency (Goforth, Primeau, & Fernandez, 2003) and are characterized by visual, auditory, and or tactile hallucinations, gross tremor, tachycardia, sweating, and, possibly, fever, as well as the disturbances of consciousness described earlier.
Tolerance develops to many effects of cannabis, including the high with chronic use, and an abstinence syndrome has been described with disturbed sleep, decreased appetite, restlessness, irritability, and sweating. Withdrawal symptoms are usually mild and short-lived, although they may be more severe in heavy regular users (82).
Though GH exerts a lipolytic effect, patients affected by acromegaly generally do not show significant weight loss. However, a decreased fat mass has been reported 39 . The most common somatic findings include acral growth (gigantism if acromegaly has a prepubertal onset), facial changes, voice reduction, arthralgias, excessive sweating, weakness, and malocclusion. Hypertension, reduced glucose tolerance or diabetes mellitus, and dyslipidaemia (hyper-trygliceridaemia) commonly occur. Although GH exerts an anabolic effect on muscle, patients with acromegaly often suffer from myopathy, with muscular weakness 36 .
The relative toxicity of ryania to mammals differs greatly between crude preparations of insecticidal dusts and the more purified extracts of the active ryanoids themselves. Ware (1988) notes that ryania's LD50 values in rats are 750 and 4000 mg kg, respectively, reflecting relatively low mammalian toxicity. However, ryanodine and 9,21-dehydroryanodine are much more toxic to mammals (Pessah et al. 1985 Bloomquist 1996). Human ingestion of large doses of ryania causes weakness, deep and slow respiration, vomiting, diarrhea, and tremors convulsions and coma precede death in fatal doses. Exposure to the more potent ryanoids in extracts causes symptoms similar to those of organophosphate poisoning. (Depending on exposure, orga-nophosphate poisoning symptoms may include sweating, headache, twitching, muscle cramps, confusion, tightness in the chest, blurred vision, vomiting, evacuation of the bowels and bladder, convulsions, respiratory failure, coma, and death.)
Phencyclidine, known as angel dust, is usually smoked, although it can be taken orally, intravenously, or by nasal inhalation. It is commonly used as an additive to other drugs, such as cannabis or LSD, and the symptoms and signs may vary greatly (49). At low doses, euphoria, relaxation, and an altered body image may occur, but at higher doses, there may be agitation, bizarre behavior, and a paranoid psychosis (50). Analgesia occurs, which may lead to self-injury. Physical effects include nystagmus (lateral and vertical), and with severe intoxication there is adrenergic stimulation with hypertension, tachycardia, flushing, hyperthermia, and cholinomimetic stimulation with sweating, hypersalivation, miosis, dystonia, ataxia, and myoclonus eventually resulting in coma, respiratory arrest, and circulatory collapse (51). Death may also result from intoxication or from violent behavior. Chronic effects of phen-cyclidine abuse include memory impairment, personality changes, and depression...
Water is a critical nutrient for the athlete in training and competition. Working muscles produce heat, and water is lost during exercise as the body attempts to keep cool and dissipates heat through sweating. Ninety minutes of strenuous exercise in a 70-kg athlete will produce sweat water losses of 1.5-3.0 kg, depending on air temperature and hu-midity.5 lost, heavy sweating for periods of up to 2-3 hours has no significant effects on electrolyte concentrations in the body.5 Although salt (sodium chloride) tablets are often promoted for athletes, only in ultra-long endurance events do electrolyte losses in sweat become significant. A single post-exercise meal replaces all the electrolytes lost in moderate exercise.
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